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Any process that stops, prevents, or reduces the rate of mast cell degranulation. The change in morphology and behavior of a mast cell resulting from exposure to a cytokine, chemokine, soluble factor, or to (at least in mammals) an antigen which the mast cell has specifically bound via IgE bound to Fc-epsilonRI receptors. The change in morphology and behavior of a mast cell resulting from exposure to a cytokine, chemokine, soluble factor, or to (at least in mammals) an antigen which the mast cell has specifically bound via IgE bound to Fc-epsilonRI receptors, leading to the initiation or perpetuation of an immune response. Any process that modulates the frequency, rate or extent of the immune response, the immunological reaction of an organism to an immunogenic stimulus. A change in the morphology or behavior of a myeloid cell resulting from exposure to an activating factor such as a cellular or soluble ligand, leading to the initiation or perpetuation of an immune response. Any process that stops, prevents, or reduces the frequency, rate, or extent of leukocyte activation. Any process that stops, prevents or reduces the frequency, rate, or extent of mast cell activation during an immune response. Any process that modulates the frequency, rate, or extent of mast cell activation during an immune response. Any process that stops, prevents or reduces the frequency, rate, or extent of mast cell activation. Any process that modulates the frequency, rate, or extent of mast cell activation.

View Gene Ontology (GO) Term

GO TERM SUMMARY
Name: negative regulation of mast cell activation during immune response
Acc: GO:0033007
Aspect: Biological Process
Desc: Any process that stops, prevents or reduces the frequency, rate, or extent of mast cell activation during an immune response.
Proteins in PDR annotated with:
   This term: 0
   Term or descendants: 2 [Search]


[geneontology.org]
INTERACTIVE GO GRAPH

GO:0033007 - negative regulation of mast cell activation during immune response (interactive image map)
YRC Informatics Platform - Version 3.0
Created and Maintained by: Michael Riffle