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Any process that stops, prevents or reduces the frequency, rate or extent of a signaling pathway. Any process that modulates the frequency, rate or extent of the cytokine mediated signaling pathway. Any process that stops, prevents or reduces the frequency, rate or extent of the cytokine mediated signaling pathway. Any series of molecular signals generated as a consequence of a cytokine or chemokine binding to a cell surface receptor. Any process that modulates the rate or extent of the tumor necrosis factor-mediated signaling pathway. The tumor necrosis factor-mediated signaling pathway is the series of molecular signals generated as a consequence of tumor necrosis factor binding to a cell surface receptor. Any process that decreases the rate or extent of the tumor necrosis factor-mediated signaling pathway. The tumor necrosis factor-mediated signaling pathway is the series of molecular signals generated as a consequence of tumor necrosis factor binding to a cell surface receptor. A series of molecular signals generated as a consequence of tumor necrosis factor binding to a cell surface receptor.

View Gene Ontology (GO) Term

GO TERM SUMMARY
Name: negative regulation of tumor necrosis factor-mediated signaling pathway
Acc: GO:0010804
Aspect: Biological Process
Desc: Any process that decreases the rate or extent of the tumor necrosis factor-mediated signaling pathway. The tumor necrosis factor-mediated signaling pathway is the series of molecular signals generated as a consequence of tumor necrosis factor binding to a cell surface receptor.
Synonyms:
  • negative regulation of tumor necrosis factor-mediated signalling pathway
  • negative regulation of TNF signaling
  • negative regulation of TNF-mediated signaling pathway
Proteins in PDR annotated with:
   This term: 1 [Search]
   Term or descendants: 1 [Search]


[geneontology.org]
INTERACTIVE GO GRAPH

GO:0010804 - negative regulation of tumor necrosis factor-mediated signaling pathway (interactive image map)
YRC Informatics Platform - Version 3.0
Created and Maintained by: Michael Riffle