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Any process that activates or increases the frequency, rate or extent of the chemical reactions and pathways by which individual cells transform chemical substances. Any process that activates or increases the frequency, rate or extent of the chemical reactions and pathways resulting in the breakdown of substances, carried out by individual cells. The process by which cells digest parts of their own cytoplasm; allows for both recycling of macromolecular constituents under conditions of cellular stress and remodeling the intracellular structure for cell differentiation. Any process that modulates the frequency, rate or extent of autophagy. Autophagy is the process by which cells digest parts of their own cytoplasm. The chemical reactions and pathways resulting in the breakdown of substances, carried out by individual cells. Any process, such as recognition of nutrient depletion, that activates or increases the rate of macroautophagy to bring cytosolic macromolecules to the vacuole/lysosome for degradation. Any process that activates or increases the frequency, rate or extent of the chemical reactions and pathways resulting in the breakdown of substances. Any process that modulates the frequency, rate or extent of the chemical reactions and pathways resulting in the breakdown of substances, carried out by individual cells. Any process that activates, maintains or increases the rate of autophagy. Autophagy is the process by which cells digest parts of their own cytoplasm.

View Gene Ontology (GO) Term

GO TERM SUMMARY
Name: positive regulation of autophagy
Acc: GO:0010508
Aspect: Biological Process
Desc: Any process that activates, maintains or increases the rate of autophagy. Autophagy is the process by which cells digest parts of their own cytoplasm.
Proteins in PDR annotated with:
   This term: 4 [Search]
   Term or descendants: 6 [Search]


[geneontology.org]
INTERACTIVE GO GRAPH

GO:0010508 - positive regulation of autophagy (interactive image map)
YRC Informatics Platform - Version 3.0
Created and Maintained by: Michael Riffle