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The directed movement of the hexose monosaccharide glucose into, out of, within or between cells by means of some external agent such as a transporter or pore. Any process that modulates the frequency, rate or extent of the import of the hexose monosaccharide glucose into a cell or organelle. The directed movement of the hexose monosaccharide glucose into a cell or organelle. Any process that stops, prevents or reduces the frequency, rate or extent of the import of the hexose monosaccharide glucose into a cell or organelle. Any process that decreases the frequency, rate or extent of glucose transport. Glucose transport is the directed movement of the hexose monosaccharide glucose into, out of, within or between cells by means of some external agent such as a transporter or pore. Any process that modulates the frequency, rate or extent of glucose transport. Glucose transport is the directed movement of the hexose monosaccharide glucose into, out of, within or between cells by means of some external agent such as a transporter or pore. Any process that stops, prevents or reduces the frequency, rate or extent of the directed movement of substances (such as macromolecules, small molecules, ions) into, out of, within or between cells by means of some external agent such as a transporter or pore.

View Gene Ontology (GO) Term

GO TERM SUMMARY
Name: negative regulation of glucose import
Acc: GO:0046325
Aspect: Biological Process
Desc: Any process that stops, prevents or reduces the frequency, rate or extent of the import of the hexose monosaccharide glucose into a cell or organelle.
Synonyms:
  • down regulation of glucose import
  • downregulation of glucose import
  • inhibition of glucose import
  • negative regulation of glucose uptake
  • down-regulation of glucose import
Proteins in PDR annotated with:
   This term: 7 [Search]
   Term or descendants: 7 [Search]


[geneontology.org]
INTERACTIVE GO GRAPH

GO:0046325 - negative regulation of glucose import (interactive image map)
YRC Informatics Platform - Version 3.0
Created and Maintained by: Michael Riffle