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Any process that stops, prevents or reduces the frequency, rate or extent of the series of molecular signals initiated by binding of a Wnt protein to a receptor on the surface of the target cell where activated receptors leads to an increase in intracellular calcium and activation of protein kinase C (PKC). Any process that modulates the frequency, rate or extent of the series of molecular signals initiated by binding of a Wnt protein to a receptor on the surface of the target cell where activated receptors leads to an increase in intracellular calcium and activation of protein kinase C (PKC). Any process that activates or increases the frequency, rate or extent of the frizzled-2 signaling pathway. The series of molecular signals initiated by binding of Wnt protein to a frizzled family receptor on the surface of the target cell and ending with a change in cell state. Any series of molecular signals initiated by the binding of an extracellular ligand to a receptor on the surface of the target cell. The series of molecular signals initiated by binding of a Wnt protein to a receptor on the surface of the target cell where activated receptors leads to an increase in intracellular calcium and activation of protein kinase C (PKC).

View Gene Ontology (GO) Term

GO TERM SUMMARY
Name: Wnt receptor signaling pathway, calcium modulating pathway
Acc: GO:0007223
Aspect: Biological Process
Desc: The series of molecular signals initiated by binding of a Wnt protein to a receptor on the surface of the target cell where activated receptors leads to an increase in intracellular calcium and activation of protein kinase C (PKC).
Synonyms:
  • non-canonical Wnt signaling pathway
  • frizzled-2 signalling pathway
  • frizzled-2 signaling pathway
Proteins in PDR annotated with:
   This term: 78 [Search]
   Term or descendants: 79 [Search]


[geneontology.org]
INTERACTIVE GO GRAPH

GO:0007223 - Wnt receptor signaling pathway, calcium modulating pathway (interactive image map)
YRC Informatics Platform - Version 3.0
Created and Maintained by: Michael Riffle